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**Õ ¼±ÐÔÒÈÏÙÑ×ÓëÈ«ÉíÐÔÑ×Ö¢·´Ó¦×ÛºÏÕ÷ 1.Deoxycholic acid inhibited proliferation and induced apoptosis and necrosis byregulating the activity of transcription factors in rat pancreatic acinar cell lineAR42J(ÍÑÑõµ¨Ëáͨ¹ýµ÷½Úת¼Òò×ӵĻîÐÔÒÖÖÆ´óÊóÒÈÏÙÏÙÅÝϸ°ûϵAR42JµÄÔöÖ³²¢ÓÕµ¼ÆäµòÍöºÍ»µËÀ) 2.Regulatory effects of apoptosis-associated speck-like protein on cytokines in theP388D1 macrophage-like cell line(µòÍöÏà¹Ø°ßµãÑùµ°°×¶Ô P388D1¾ÞÊÉϸ°ûÑùϸ°ûϵϸ°ûÒò×ӵĵ÷¿Ø×÷ÓÃ) 3.CEBPE Promotes LPS-Induced IL-1B Transcription and Secretion in AlveolarMacrophages via NOD2 Signaling (CEBPB´Ù½øLPSÓÕµ¼µÄ·ÎÅݾÞÊÉϸ°ûIL-1pµÄת¼ºÍ·ÖÃÚ) 4.Identification of Key Biomarkers Associated with Immunogenic Cell Death andTheir Regulatory Mechanisms in Severe Acute Pancreatitis Based on WGCNA andMachine Learning(»ùÓÚWGCNA ºÍ»úÆ÷ѧϰ·½·¨¶ÔÖØÖ¢¼±ÐÔÒÈÏÙÑ×ʱÃâÒßÔ´ÐÔϸ°ûËÀÍö¼° Æäµ÷½Ú»úÖÆÏà¹ØµÄ¹Ø¼üÉúÎï±êÖ¾ÎïµÄÈ·¶¨) 5.Exosome-derived CIRP: An amplifier of inflammatory diseases(ÍâÃÚÌåÌØÒìÐÔCIRP;Ñ×Ö¢·´Ó¦µÄ·Å´óÆ÷) 6.Protective Effects of Emodin-Induced Neutrophil Apoptosis via the Ca*-Caspase12 Pathway against SIRS in Rats with Severe Acute Pancreatitis (´ó»ÆËØÓÕµ¼µÄÖÐÐÔÁ£Ï¸°ûµòÍöͨ¹ýCa*-Caspase 12;¾¶¶ÔÖØÖ¢¼±ÐÔÒÈÏÙÑ×´óÊóµÄSIRS ¾ßÓб£»¤×÷ÓÃ) 7.Inhibition of Necroptosis in Acute Pancreatitis: Screening for RIPK1 Inhibitors(¼±ÒÈÏÙÑ×»µËÀÐÔµòÍöµÄÒÖÖÆ:RIPK1ÒÖÖƼÁµÄɸѡ) 8.Regulatory effects of emodin on NF-kB activation and infammatory cytokine expressiorin RAW 264.7 macrophages (´ó»ÆËØ¶Ô RAW 264.7 ¾ÞÊÉϸ°ûÖÐNF-xB»î»¯ºÍÑ×Ö¢Òò×Ó±í´ïµÄµ÷½Ú×÷ÓÃ) 9.Qingyi Deces acute biliary pancreatitis by targeting Gpbar1NF-rb pathway (ÇåÒÈÌÀ°ÐÏòGpbar1NF-rbͨ·»º½â¼±ÐÔµ¨Ô´ÐÔÒÈÏÙÑ×) 10.Effects of Qingyi decoction onammatory markers in patients with acute pancreatitis: Aeta-analysis (ÇåÒÈÌÀ¶Ô¼±ÐÔÒÈÏÙÑ×»¼ÕßÑ× ÐÔ±êÖ¾ÎïµÄÓ°Ïì:Ü¿ÝÍ·ÖÎö) µÚ¶þÕ ¼±ÐÔÒÈÏÙÑ׳¦µÀÆÁÕÏËðÉË 1.Intestinal barrier damage, systemic inflammatory response syndrome, and acute lung in-jury: A troublesome trio for acute pancreatitis(³¦µÀÆÁÕÏËðÉË¡¢È«ÉíÐÔÑ×Ö¢·´Ó¦×ÛºÏÕ÷ºÍ·ÎËðÉË:¼±ÐÔÒÈÏÙÑ×µÄÈýÖØ×à) 2.The gut-lung axis in severe acute Pancreatitis-associated lung injury: The protection bythe gut microbiota through short-chain fatty acids(³¦µÀ¾úȺͨ¹ý¶ÌÁ´Ö¬·¾Ëá¶Ô¼±ÐÔÒÈÏÙÑ×·ÎËðÉËʱ³¦-·ÎÖáµÄ±£»¤×÷ÓÃ) 3.Intestinal Microbiota - An Unmissable Bridge to Severe Acute Pancreatitis-AssociatedAcute Lung Injury (³¦µÀ¾úȺ¶Ô¼±ÐÔÒÈÏÙÑ× Ïà¹ØÐÔ·ÎËðÉ˾ßÓв»¿É»òȱµÄ×÷ÓÃ) 4.Therapeutic effect of Qingyi decoction in severe acute pancreatitis-induced intestinal bar-rier injury(ÇåÒÈÌÀ¶ÔÖØÖ¢¼±ÐÔÒÈÏÙÑ×ËùÖ³¦µÀÆÁÕÏËðÉ˵ÄÖÎÁÆ×÷ÓÃ) 5.Qingyi decoction attenuates intestinal epithelial cell injury via the calcineurinnuclearfactor of activated T-cells pathway(ÇåÒÈÌÀ ͨ¹ýNFAT ϸ°ûͨ·¼õ»º³¦ÉÏƤϸ°ûËðÉË) µÚÈýÕ ¼±ÐÔÒÈÏÙÑ×·ÎËðÉË 1.Effects and mechanisms of alveolar type Il epithelial cell apoptosis in severe pancreatitis-induced acute lung injury (·ÎÅÝIÐÍÉÏƤϸ °ûµòÍöÔÚÖØÖ¢¼±ÐÔÒÈÏÙÑ×ËùÖ¼±ÐÔ·ÎËðÉËÖеÄ×÷ÓúͻúÖÆ) 2.Enhancement of ICAM-I via the JAK2STAT3 signaling pathway in a rat model of severeacute pancreatitis-associated lung injury (JAK2STAT3 ÐźÅͨ·ÔÚÖØÖ¢¼±ÐÔÒÈÏÙÑ×Ïà¹Ø·ÎËðÉË´óÊóÄ£ÐÍÖжÔICAM-IµÄÔöǿЧӦ) 3.Inhibition of SOCs Attenuates Acute Lung Injury Induced by Severe Acute Pancreatitis inRats and PMVECs Injury Induced by Lipopolysaccharide(ÒÖÖƸƿâ²Ù×ÝÐÔ¸ÆÀë×ÓͨµÀ¿É¼õÇá´óÊóÖØÖ¢¼±ÐÔÒÈÏÙÑ×·ÎËðÉ˺ÍÖ¬¶àÌÇËùÖ·ÎѪ¹ÜÄÚƤϸ°ûËðÉË) Effect of angiopoietin-like protein 4 on rat pulmonary microvascular endothelial cells ex-posed to LPS(Ѫ¹ÜÉú³ÉËØÑùµ°°×4¶Ô LPS ËùÖ´óÊó·Î΢Ѫ¹ÜÄÚƤϸ°ûµÄÓ°Ïì) 4.Effects of hepoxia-inlucible factor-lor andatrix metalloproteinase-9 on alveolar-capillary barrier disru ung edemaodels of severe acute pancreatitis associated lung injury (ȱÑõÓÕµ¼Òò×Ó-1cºÍ»ùÖʽðÊôµ°°×ø-9¶ÔÖØÖ¢¼±ÐÔÒÈÏÙÑ×Ïà¹Ø·ÎËðÉË´óÊó·ÎÅÝ-ëϸѪ¹ÜÆÁÕÏÆÆ»µºÍ ·ÎË®Ö×µÄÓ°Ïì) 6.Multifunctional neuron-specific enolase: its ro